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Genetic polymorphisms of pharmacogenomic VIP variants in Li nationality of southern China.

Environmental toxicology and pharmacology

Ding Y, He P, He N, Li Q, Sun J, Yao J, Yi S, Xu H, Wu D, Wang X, Jin T.
PMID: 26901752
Environ Toxicol Pharmacol. 2016 Mar;42:237-42. doi: 10.1016/j.etap.2016.02.003. Epub 2016 Feb 20.

OBJECTIVES: The present study aimed to screen members of the Li nationality in southern China for genotype frequencies of VIP variants and to determine differences between the Li ethnicity and global human population samples in HapMap.METHODS: In this study,...

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Ding Y, He P, He N, et al. Genetic polymorphisms of pharmacogenomic VIP variants in Li nationality of southern China. Environ Toxicol Pharmacol. 2016;42:237-42doi: 10.1016/j.etap.2016.02.003.
Ding, Y., He, P., He, N., Li, Q., Sun, J., Yao, J., Yi, S., Xu, H., Wu, D., Wang, X., & Jin, T. (2016). Genetic polymorphisms of pharmacogenomic VIP variants in Li nationality of southern China. Environmental toxicology and pharmacology, 42237-42. https://doi.org/10.1016/j.etap.2016.02.003
Ding, Yipeng, et al. "Genetic polymorphisms of pharmacogenomic VIP variants in Li nationality of southern China." Environmental toxicology and pharmacology vol. 42 (2016): 237-42. doi: https://doi.org/10.1016/j.etap.2016.02.003
Ding Y, He P, He N, Li Q, Sun J, Yao J, Yi S, Xu H, Wu D, Wang X, Jin T. Genetic polymorphisms of pharmacogenomic VIP variants in Li nationality of southern China. Environ Toxicol Pharmacol. 2016 Mar;42:237-42. doi: 10.1016/j.etap.2016.02.003. Epub 2016 Feb 20. PMID: 26901752.
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Potential biomarkers of trichloroethylene and toluene exposure in Corbicula fluminea.

Environmental toxicology and pharmacology

Vidal M, Bassères A, Narbonne J.
PMID: 11167153
Environ Toxicol Pharmacol. 2001 Jan 01;9(3):87-97. doi: 10.1016/s1382-6689(00)00068-5.

Freshwater clams Corbicula fluminea were exposed in aquariums to four doses of trichloroethylene-TCE-(1.56 up to 100 mg/1) or toluene-TOL-(7.5 up to 60 mg/1) for 5 days. At the end of exposure, components of (de)toxification metabolism of phases I and...

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Vidal M, Bassères A, Narbonne J. Potential biomarkers of trichloroethylene and toluene exposure in Corbicula fluminea. Environ Toxicol Pharmacol. 2001;9(3):87-97doi: 10.1016/s1382-6689(00)00068-5.
Vidal, M., Bassères, A., & Narbonne, J. (2001). Potential biomarkers of trichloroethylene and toluene exposure in Corbicula fluminea. Environmental toxicology and pharmacology, 9(3), 87-97. https://doi.org/10.1016/s1382-6689(00)00068-5
Vidal, M -L., et al. "Potential biomarkers of trichloroethylene and toluene exposure in Corbicula fluminea." Environmental toxicology and pharmacology vol. 9,3 (2001): 87-97. doi: https://doi.org/10.1016/s1382-6689(00)00068-5
Vidal M, Bassères A, Narbonne J. Potential biomarkers of trichloroethylene and toluene exposure in Corbicula fluminea. Environ Toxicol Pharmacol. 2001 Jan 01;9(3):87-97. doi: 10.1016/s1382-6689(00)00068-5. PMID: 11167153.
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Characterization of low dose streptozotocin-induced progressive diabetes in mice.

Environmental toxicology and pharmacology

Ito M, Kondo Y, Nakatani A, Hayashi K, Naruse A.
PMID: 11167151
Environ Toxicol Pharmacol. 2001 Jan 01;9(3):71-78. doi: 10.1016/s1382-6689(00)00064-8.

Our previous study indicated that a single i.p. injection of 100 mg/kg streptozotocin (STZ) is able to induce slowly progressive diabetes mellitus in adult ICR mice. In the present study, the non-fasting serum insulin levels of the mice administered...

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Ito M, Kondo Y, Nakatani A, et al. Characterization of low dose streptozotocin-induced progressive diabetes in mice. Environ Toxicol Pharmacol. 2001;9(3):71-78doi: 10.1016/s1382-6689(00)00064-8.
Ito, M., Kondo, Y., Nakatani, A., Hayashi, K., & Naruse, A. (2001). Characterization of low dose streptozotocin-induced progressive diabetes in mice. Environmental toxicology and pharmacology, 9(3), 71-78. https://doi.org/10.1016/s1382-6689(00)00064-8
Ito, M, et al. "Characterization of low dose streptozotocin-induced progressive diabetes in mice." Environmental toxicology and pharmacology vol. 9,3 (2001): 71-78. doi: https://doi.org/10.1016/s1382-6689(00)00064-8
Ito M, Kondo Y, Nakatani A, Hayashi K, Naruse A. Characterization of low dose streptozotocin-induced progressive diabetes in mice. Environ Toxicol Pharmacol. 2001 Jan 01;9(3):71-78. doi: 10.1016/s1382-6689(00)00064-8. PMID: 11167151.
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Inflammatory responses in RAW264.7 macrophages caused by mycobacteria isolated from moldy houses.

Environmental toxicology and pharmacology

Huttunen K, Ruotsalainen M, Iivanainen E, Torkko P, Katila M, Hirvonen M.
PMID: 10996543
Environ Toxicol Pharmacol. 2000 Jun 01;8(4):237-244. doi: 10.1016/s1382-6689(00)00047-8.

Mycobacterial strains (nonpathogenic Mycobacterium terrae, potentially pathogenic Mycobacterium avium-complex and Mycobacterium scrofulaceum), isolated from a moldy building, were studied with respect to their ability to stimulate macrophages (RAW264.7) to produce inflammatory mediators, and to cause cytotoxicity. Reactive oxygen species...

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Huttunen K, Ruotsalainen M, Iivanainen E, et al. Inflammatory responses in RAW264.7 macrophages caused by mycobacteria isolated from moldy houses. Environ Toxicol Pharmacol. 2000;8(4):237-244doi: 10.1016/s1382-6689(00)00047-8.
Huttunen, K., Ruotsalainen, M., Iivanainen, E., Torkko, P., Katila, M., & Hirvonen, M. (2000). Inflammatory responses in RAW264.7 macrophages caused by mycobacteria isolated from moldy houses. Environmental toxicology and pharmacology, 8(4), 237-244. https://doi.org/10.1016/s1382-6689(00)00047-8
Huttunen, et al. "Inflammatory responses in RAW264.7 macrophages caused by mycobacteria isolated from moldy houses." Environmental toxicology and pharmacology vol. 8,4 (2000): 237-244. doi: https://doi.org/10.1016/s1382-6689(00)00047-8
Huttunen K, Ruotsalainen M, Iivanainen E, Torkko P, Katila M, Hirvonen M. Inflammatory responses in RAW264.7 macrophages caused by mycobacteria isolated from moldy houses. Environ Toxicol Pharmacol. 2000 Jun 01;8(4):237-244. doi: 10.1016/s1382-6689(00)00047-8. PMID: 10996543.
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Evidence of modified LDL in the plasma of hypercholesterolemic WHHL rabbits injected with aqueous extracts of cigarette smoke.

Environmental toxicology and pharmacology

Yamaguchi Y, Kagota S, Haginaka J, Kunitomo M.
PMID: 10996545
Environ Toxicol Pharmacol. 2000 Jun 01;8(4):255-260. doi: 10.1016/s1382-6689(00)00050-8.

Cigarette smoking is a major risk factor for atherosclerosis, and oxidative modification of low density lipoprotein (LDL) is a key event in the development of atherosclerosis. The aim of the present study was to determine whether modified LDL would...

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Yamaguchi Y, Kagota S, Haginaka J, et al. Evidence of modified LDL in the plasma of hypercholesterolemic WHHL rabbits injected with aqueous extracts of cigarette smoke. Environ Toxicol Pharmacol. 2000;8(4):255-260doi: 10.1016/s1382-6689(00)00050-8.
Yamaguchi, Y., Kagota, S., Haginaka, J., & Kunitomo, M. (2000). Evidence of modified LDL in the plasma of hypercholesterolemic WHHL rabbits injected with aqueous extracts of cigarette smoke. Environmental toxicology and pharmacology, 8(4), 255-260. https://doi.org/10.1016/s1382-6689(00)00050-8
Yamaguchi, et al. "Evidence of modified LDL in the plasma of hypercholesterolemic WHHL rabbits injected with aqueous extracts of cigarette smoke." Environmental toxicology and pharmacology vol. 8,4 (2000): 255-260. doi: https://doi.org/10.1016/s1382-6689(00)00050-8
Yamaguchi Y, Kagota S, Haginaka J, Kunitomo M. Evidence of modified LDL in the plasma of hypercholesterolemic WHHL rabbits injected with aqueous extracts of cigarette smoke. Environ Toxicol Pharmacol. 2000 Jun 01;8(4):255-260. doi: 10.1016/s1382-6689(00)00050-8. PMID: 10996545.
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Preliminary in vitro toxicological evaluation of a series of 2-pyridylcarboxamidrazone candidate anti-tuberculosis compounds: II(1).

Environmental toxicology and pharmacology

Coleman MD, Rathbone DL, Endersby CR, Hovey MC, Tims KJ, Lambert PA, Billington DC.
PMID: 10925069
Environ Toxicol Pharmacol. 2000 Mar 01;8(3):167-172. doi: 10.1016/s1382-6689(00)00037-5.

The in vitro toxicity of two amidrazones I [N(1)-(3-benzyloxy-4-methoxybenzylidene)-pyridine-2-carboxamidrazone] and II [N(1)-(4-benzyloxy-3-methoxybenzylidene)-pyridine-2-carboxamidrazone] and their precursors PI (3-benzyloxy-4-methoxybenzaldehyde) and PII (4-benzyloxy-3-methoxybenzaldehyde) was determined using a rat liver metabolism system with human mononuclear leucocytes (MNL) as target cells. The minimum inhibitory...

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Coleman MD, Rathbone DL, Endersby CR, et al. Preliminary in vitro toxicological evaluation of a series of 2-pyridylcarboxamidrazone candidate anti-tuberculosis compounds: II(1). Environ Toxicol Pharmacol. 2000;8(3):167-172doi: 10.1016/s1382-6689(00)00037-5.
Coleman, M. D., Rathbone, D. L., Endersby, C. R., Hovey, M. C., Tims, K. J., Lambert, P. A., & Billington, D. C. (2000). Preliminary in vitro toxicological evaluation of a series of 2-pyridylcarboxamidrazone candidate anti-tuberculosis compounds: II(1). Environmental toxicology and pharmacology, 8(3), 167-172. https://doi.org/10.1016/s1382-6689(00)00037-5
Coleman, et al. "Preliminary in vitro toxicological evaluation of a series of 2-pyridylcarboxamidrazone candidate anti-tuberculosis compounds: II(1)." Environmental toxicology and pharmacology vol. 8,3 (2000): 167-172. doi: https://doi.org/10.1016/s1382-6689(00)00037-5
Coleman MD, Rathbone DL, Endersby CR, Hovey MC, Tims KJ, Lambert PA, Billington DC. Preliminary in vitro toxicological evaluation of a series of 2-pyridylcarboxamidrazone candidate anti-tuberculosis compounds: II(1). Environ Toxicol Pharmacol. 2000 Mar 01;8(3):167-172. doi: 10.1016/s1382-6689(00)00037-5. PMID: 10925069.
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Deltamethrin induces delayed apoptosis and altered expression of p53 and bax in rat brain.

Environmental toxicology and pharmacology

Wu A, Liu Y.
PMID: 10925071
Environ Toxicol Pharmacol. 2000 Mar 01;8(3):183-189. doi: 10.1016/s1382-6689(00)00039-9.

Our previous work indicates that deltamethrin induces degeneration and apoptosis in rat brain at 24 and 48 h after treatment. To determine whether molecular characteristics of apoptosis is involved in neurodegeneration in rat brain after deltamethrin treatment, we investigated...

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Wu A, Liu Y. Deltamethrin induces delayed apoptosis and altered expression of p53 and bax in rat brain. Environ Toxicol Pharmacol. 2000;8(3):183-189doi: 10.1016/s1382-6689(00)00039-9.
Wu, A., & Liu, Y. (2000). Deltamethrin induces delayed apoptosis and altered expression of p53 and bax in rat brain. Environmental toxicology and pharmacology, 8(3), 183-189. https://doi.org/10.1016/s1382-6689(00)00039-9
Wu, and Liu. "Deltamethrin induces delayed apoptosis and altered expression of p53 and bax in rat brain." Environmental toxicology and pharmacology vol. 8,3 (2000): 183-189. doi: https://doi.org/10.1016/s1382-6689(00)00039-9
Wu A, Liu Y. Deltamethrin induces delayed apoptosis and altered expression of p53 and bax in rat brain. Environ Toxicol Pharmacol. 2000 Mar 01;8(3):183-189. doi: 10.1016/s1382-6689(00)00039-9. PMID: 10925071.
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Pharmacological property of tributyltin in vivo and in vitro.

Environmental toxicology and pharmacology

Mizuhashi S, Ikegaya Y, Matsuki N.
PMID: 10925074
Environ Toxicol Pharmacol. 2000 Mar 01;8(3):205-212. doi: 10.1016/s1382-6689(00)00042-9.

Tributyltin (TBT), an assumed endocrine-disrupting chemical, is widely known to show harmful effects in invertebrates including the dioecious snails. As for mammals, there are several reports concerning TBT toxicology, but few indications about general pharmacology of TBT. In the...

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Mizuhashi S, Ikegaya Y, Matsuki N. Pharmacological property of tributyltin in vivo and in vitro. Environ Toxicol Pharmacol. 2000;8(3):205-212doi: 10.1016/s1382-6689(00)00042-9.
Mizuhashi, S., Ikegaya, Y., & Matsuki, N. (2000). Pharmacological property of tributyltin in vivo and in vitro. Environmental toxicology and pharmacology, 8(3), 205-212. https://doi.org/10.1016/s1382-6689(00)00042-9
Mizuhashi, et al. "Pharmacological property of tributyltin in vivo and in vitro." Environmental toxicology and pharmacology vol. 8,3 (2000): 205-212. doi: https://doi.org/10.1016/s1382-6689(00)00042-9
Mizuhashi S, Ikegaya Y, Matsuki N. Pharmacological property of tributyltin in vivo and in vitro. Environ Toxicol Pharmacol. 2000 Mar 01;8(3):205-212. doi: 10.1016/s1382-6689(00)00042-9. PMID: 10925074.
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Dinoflagellates from marine algal blooms produce neurotoxic compounds: effects on free calcium levels in neuronal cells and synaptosomes.

Environmental toxicology and pharmacology

Perovic S, Tretter L, Brümmer F, Wetzler C, Brenner J, Donner G, Schröder HC, Müller WE.
PMID: 10867367
Environ Toxicol Pharmacol. 2000 Jan 01;8(2):83-94. doi: 10.1016/s1382-6689(99)00035-6.

In this report, evidence is presented that the marine unicellular eukaryotic dinoflagellates can cause neurotoxicity very likely by an increase in intracellular free calcium ions ([Ca(2+)](i)). Determinations of the effects of culture supernatants from different clones of the dinoflagellate...

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Perovic S, Tretter L, Brümmer F, et al. Dinoflagellates from marine algal blooms produce neurotoxic compounds: effects on free calcium levels in neuronal cells and synaptosomes. Environ Toxicol Pharmacol. 2000;8(2):83-94doi: 10.1016/s1382-6689(99)00035-6.
Perovic, S., Tretter, L., Brümmer, F., Wetzler, C., Brenner, J., Donner, G., Schröder, H. C., & Müller, W. E. (2000). Dinoflagellates from marine algal blooms produce neurotoxic compounds: effects on free calcium levels in neuronal cells and synaptosomes. Environmental toxicology and pharmacology, 8(2), 83-94. https://doi.org/10.1016/s1382-6689(99)00035-6
Perovic, et al. "Dinoflagellates from marine algal blooms produce neurotoxic compounds: effects on free calcium levels in neuronal cells and synaptosomes." Environmental toxicology and pharmacology vol. 8,2 (2000): 83-94. doi: https://doi.org/10.1016/s1382-6689(99)00035-6
Perovic S, Tretter L, Brümmer F, Wetzler C, Brenner J, Donner G, Schröder HC, Müller WE. Dinoflagellates from marine algal blooms produce neurotoxic compounds: effects on free calcium levels in neuronal cells and synaptosomes. Environ Toxicol Pharmacol. 2000 Jan 01;8(2):83-94. doi: 10.1016/s1382-6689(99)00035-6. PMID: 10867367.
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Comparative characterization of CHCl(3) metabolism and toxicokinetics in rodent strains differently susceptible to chloroform-induced carcinogenicity.

Environmental toxicology and pharmacology

Vittozzi L, Gemma S, Sbraccia M, Testai E.
PMID: 10867369
Environ Toxicol Pharmacol. 2000 Jan 01;8(2):103-110. doi: 10.1016/s1382-6689(00)00031-4.

A comparative kinetic study in B6C3F1 mice, Osborne-Mendel (OM) and Sprague-Dawley (SD) rats has been undertaken with the major aim to determine the extent of covalent binding of chloroform reactive metabolites produced in vivo through oxidative and/or reductive metabolism...

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Vittozzi L, Gemma S, Sbraccia M, et al. Comparative characterization of CHCl(3) metabolism and toxicokinetics in rodent strains differently susceptible to chloroform-induced carcinogenicity. Environ Toxicol Pharmacol. 2000;8(2):103-110doi: 10.1016/s1382-6689(00)00031-4.
Vittozzi, L., Gemma, S., Sbraccia, M., & Testai, E. (2000). Comparative characterization of CHCl(3) metabolism and toxicokinetics in rodent strains differently susceptible to chloroform-induced carcinogenicity. Environmental toxicology and pharmacology, 8(2), 103-110. https://doi.org/10.1016/s1382-6689(00)00031-4
Vittozzi, et al. "Comparative characterization of CHCl(3) metabolism and toxicokinetics in rodent strains differently susceptible to chloroform-induced carcinogenicity." Environmental toxicology and pharmacology vol. 8,2 (2000): 103-110. doi: https://doi.org/10.1016/s1382-6689(00)00031-4
Vittozzi L, Gemma S, Sbraccia M, Testai E. Comparative characterization of CHCl(3) metabolism and toxicokinetics in rodent strains differently susceptible to chloroform-induced carcinogenicity. Environ Toxicol Pharmacol. 2000 Jan 01;8(2):103-110. doi: 10.1016/s1382-6689(00)00031-4. PMID: 10867369.
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Assessment of multipathway exposure of small children to PAH.

Environmental toxicology and pharmacology

Vyskocil A, Fiala Z, Chénier V, Krajak L, Ettlerova E, Bukac J, Viau C, Emminger S.
PMID: 10867370
Environ Toxicol Pharmacol. 2000 Jan 01;8(2):111-118. doi: 10.1016/s1382-6689(00)00032-6.

The aim of study was to assess the uptake of polycyclic aromatic hydrocarbons (PAH) by children living in a city and its effect on 1-hydroxypyrene (1-OHP) excretion. Two groups of children (n=11 and 13; 3-6 years old) were chosen:...

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Vyskocil A, Fiala Z, Chénier V, et al. Assessment of multipathway exposure of small children to PAH. Environ Toxicol Pharmacol. 2000;8(2):111-118doi: 10.1016/s1382-6689(00)00032-6.
Vyskocil, A., Fiala, Z., Chénier, V., Krajak, L., Ettlerova, E., Bukac, J., Viau, C., & Emminger, S. (2000). Assessment of multipathway exposure of small children to PAH. Environmental toxicology and pharmacology, 8(2), 111-118. https://doi.org/10.1016/s1382-6689(00)00032-6
Vyskocil, et al. "Assessment of multipathway exposure of small children to PAH." Environmental toxicology and pharmacology vol. 8,2 (2000): 111-118. doi: https://doi.org/10.1016/s1382-6689(00)00032-6
Vyskocil A, Fiala Z, Chénier V, Krajak L, Ettlerova E, Bukac J, Viau C, Emminger S. Assessment of multipathway exposure of small children to PAH. Environ Toxicol Pharmacol. 2000 Jan 01;8(2):111-118. doi: 10.1016/s1382-6689(00)00032-6. PMID: 10867370.
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ETAP changes.

Environmental toxicology and pharmacology

Vermeulen NP, Seegal RF, Koeman JH.
PMID: 11382557
Environ Toxicol Pharmacol. 2001 Jun;10(1):V. doi: 10.1016/s1382-6689(01)00086-2.

No abstract available.

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Vermeulen NP, Seegal RF, Koeman JH. ETAP changes. Environ Toxicol Pharmacol. 2001;10(1):Vdoi: 10.1016/s1382-6689(01)00086-2.
Vermeulen, N. P., Seegal, R. F., & Koeman, J. H. (2001). ETAP changes. Environmental toxicology and pharmacology, 10(1), V. https://doi.org/10.1016/s1382-6689(01)00086-2
Vermeulen, N P.E., et al. "ETAP changes." Environmental toxicology and pharmacology vol. 10,1 (2001): V. doi: https://doi.org/10.1016/s1382-6689(01)00086-2
Vermeulen NP, Seegal RF, Koeman JH. ETAP changes. Environ Toxicol Pharmacol. 2001 Jun;10(1):V. doi: 10.1016/s1382-6689(01)00086-2. PMID: 11382557.
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